18F-FP-(+)-DTBZ positron emission tomography detection of monoaminergic deficient network in patients with carbon monoxide related parkinsonism

Author:

Chang C.-C.1,Hsiao I.-T.23,Huang S.-H.4,Lui C.-C.5,Yen T.-C.23,Chang W.-N.1,Huang C.-W.1,Hsieh C.-J.34,Chang Y.-Y.1,Lin K.-J.23ORCID

Affiliation:

1. Department of Neurology; Kaohsiung Chang Gung Memorial Hospital; Chang Gung University College of Medicine; Kaohsiung Taiwan

2. Healthy Aging Research Centre and Department of Medical Imaging and Radiological Sciences; Chang Gung University; Taoyuan Taiwan

3. Department of Nuclear Medicine and Centre for Advanced Molecular Imaging and Translation; Chang Gung Memorial Hospital; Taoyuan Taiwan

4. Department of Nuclear Medicine; Kaohsiung Chang Gung Memorial Hospital; Chang Gung University College of Medicine; Kaohsiung Taiwan

5. Department of Radiology; Kaohsiung Chang Gung Memorial Hospital; Chang Gung University College of Medicine; Kaohsiung Taiwan

Funder

Chang Gung Memorial Hospital and National science council

Publisher

Wiley

Subject

Neurology (clinical),Neurology

Reference50 articles.

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2. The brain lesion responsible for parkinsonism after carbon monoxide poisoning;Sohn;Arch Neurol,2000

3. Primary progressive freezing gait in a patient with CO-induced parkinsonism;Lee;Mov Disord,2010

4. Pallidoreticular lesion in carbon monoxide intoxication by gradient echo: report of a case with parkinsonism features and review of the literature;Chen;Acta Neurol Taiwan,2012

5. Clinical significance of the pallidoreticular pathway in patients with carbon monoxide intoxication;Chang;Brain,2011

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