Carboxypeptidase B2 deficiency reveals opposite effects of complement C3a and C5a in a murine polymicrobial sepsis model-Reference-Cited by-同舟云学术

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Carboxypeptidase B2 deficiency reveals opposite effects of complement C3a and C5a in a murine polymicrobial sepsis model

Published:2015-05-10 Issue:6 Volume:13 Page:1090-1102
ISSN:1538-7933
Container-title:Journal of Thrombosis and Haemostasis
language:en
Short-container-title:J Thromb Haemost

Author:

Shao Z.¹²,Nishimura T.³,Leung L. L. K.¹²,Morser J.¹²

Affiliation:

1. Division of Hematology; Department of Medicine; Stanford University School of Medicine; Stanford CA USA

2. Veterans Affairs Palo Alto Health Care System; Palo Alto CA USA

3. Department of Anesthesiology; Stanford University School of Medicine; Stanford CA USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Hematology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jth.12956/fullpdf

Reference45 articles.

1. TAFI, or plasma procarboxypeptidase B, couples the coagulation and fibrinolytic cascades through the thrombin-thrombomodulin complex;Bajzar;J Biol Chem,1996

2. Thrombin activatable fibrinolysis inhibitor, a potential regulator of vascular inflammation;Myles;J Biol Chem,2003

3. Regulation of tissue inflammation by thrombin-activatable carboxypeptidase B (or TAFI);Leung;Mol Immunol,2008

4. Hemostasis and Vascular Inflammation

5. What has been learnt from the thrombin-activatable fibrinolysis inhibitor-deficient mouse?;Morser;J Thromb Haemost,2010

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