The small GTPase Arf6 is dysregulated in a mouse model for fragile X syndrome-Reference-Cited by-同舟云学术

The small GTPase Arf6 is dysregulated in a mouse model for fragile X syndrome

Published:2020-12-12 Issue:3 Volume:157 Page:666-683
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Briševac Dušica¹²,Scholz Ralf³^ORCID,Du Dan⁴,Elagabani Mohammad Nael¹^ORCID,Köhr Georg⁴⁵^ORCID,Kornau Hans‐Christian¹³⁶^ORCID

Affiliation:

1. Neuroscience Research Center (NWFZ) Charité ‐ Universitätsmedizin Berlin Berlin Germany

2. Department of Biology, Chemistry, and Pharmacy Freie Universität Berlin Berlin Germany

3. Center for Molecular Neurobiology (ZMNH) University Medical Center Hamburg‐Eppendorf (UKE) Hamburg Germany

4. Central Institute of Mental Health Mannheim Center for Translational Neuroscience, Medical Faculty Mannheim, University of Heidelberg Mannheim Germany

5. Department of Neurophysiology Mannheim Center for Translational Neuroscience, Medical Faculty Mannheim, University of Heidelberg Mannheim Germany

6. German Center for Neurodegenerative Diseases (DZNE) Berlin Berlin Germany

Funder

Deutsche Forschungsgemeinschaft

China Scholarship Council

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jnc.15230

Reference99 articles.

1. The formation of synaptic junctions in developing rat brain: A quantitative electron microscopic study

2. Altered surface mGluR5 dynamics provoke synaptic NMDAR dysfunction and cognitive defects in Fmr1 knockout mice

3. Bi-allelic Variants in IQSEC1 Cause Intellectual Disability, Developmental Delay, and Short Stature

4. Metabotropic Glutamate Receptor Activation Regulates Fragile X Mental Retardation Protein and Fmr1 mRNA Localization Differentially in Dendrites and at Synapses

5. FMRP targets distinct mRNA sequence elements to regulate protein expression

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