<scp>FasL</scp> is a catabolic factor in alveolar bone homeostasis-Reference-Cited by-同舟云学术

FasL is a catabolic factor in alveolar bone homeostasis

Published:2022-11-25 Issue:3 Volume:50 Page:396-405
ISSN:0303-6979
Container-title:Journal of Clinical Periodontology
language:en
Short-container-title:J Clinic Periodontology

Author:

Apaza Alccayhuaman Karol Alí¹²^ORCID,Heimel Patrick²³⁴,Lee Jung Seok¹⁵^ORCID,Tangl Stefan²⁴,Kuchler Ulrike⁶,Marchesan Julie⁷,Panahipour Layla¹,Lettner Stefan²⁴,Matalová Eva⁸,Gruber Reinhard¹⁴⁹^ORCID

Affiliation:

1. Department of Oral Biology University Clinic of Dentistry, Medical University of Vienna Vienna Austria

2. Karl Donath Laboratory for Hard Tissue and Biomaterial Research University Clinic of Dentistry, Medical University of Vienna Vienna Austria

3. Department for Bioimaging Ludwig Boltzmann Institute for Traumatology, The Research Center in Cooperation With AUVA Vienna Austria

4. Austrian Cluster for Tissue Regeneration Vienna Austria

5. Department of Periodontology Research Institute for Periodontal Regeneration, College of Dentistry, Yonsei University Seoul Republic of Korea

6. Department of Oral Surgery University Clinic of Dentistry, Medical University of Vienna Vienna Austria

7. Division of Comprehensive Oral Health Adams School of Dentistry, University of North Carolina at Chapel Hill Chapel Hill North Carolina USA

8. Laboratory of Odontogenesis and Osteogenesis Institute of Animal Physiology and Genetics, Czech Academy of Sciences Brno Czech Republic

9. Department of Periodontology School of Dental Medicine, University of Bern Bern Switzerland

Funder

Austrian Science Fund

Grantová Agentura České Republiky

Osteology Foundation

Publisher

Wiley

Subject

Periodontics

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcpe.13750

Reference49 articles.

1. FasL Is Required for Osseous Healing in Extraction Sockets in Mice

2. Association analysis of FAS‑670A/G and FASL‑844C/T polymorphisms with risk of generalized aggressive periodontitis disease

3. Dual role of TGF-β1 on Fas-induced apoptosis in lung epithelial cells

4. Bone development

5. Porphyromonas gingivalis enhances FasL expression via up-regulation of NFκB-mediated gene transcription and induces apoptotic cell death in human gingival epithelial cells

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