Treatment with human immunoglobulin G improves the early disease course in a mouse model of Duchenne muscular dystrophy-Reference-Cited by-同舟云学术

Treatment with human immunoglobulin G improves the early disease course in a mouse model of Duchenne muscular dystrophy

Published:2015-08-28 Issue:2 Volume:136 Page:351-362
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Zschüntzsch Jana¹,Zhang Yaxin²,Klinker Florian³,Makosch Gregor⁴,Klinge Lars⁵,Malzahn Dörthe⁶,Brinkmeier Heinrich²,Liebetanz David³,Schmidt Jens¹⁴

Affiliation:

1. Department of Neurology; University Medical Center Göttingen; Göttingen Germany

2. Institute of Pathophysiology; University Medicine Greifswald; Karlsburg Germany

3. Department of Clinical Neurophysiology; University Medical Center Göttingen; Göttingen Germany

4. Department of Neuroimmunology; Institute for Multiple Sclerosis Research and Hertie Foundation; University Medical Center Göttingen; Göttingen Germany

5. Department of Pediatrics; University Medical Center Göttingen; Göttingen Germany

6. Department of Genetic Epidemiology; University Medical Center Göttingen; Göttingen Germany

Funder

CSL Behring

Aktion Benni & Co e.V.

RHB Polyradikulitis Forschungs-GmbH

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.13269/fullpdf

Reference55 articles.

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2. Skeletal muscle fatigue: cellular mechanisms;Allen;Physiol. Rev.,2008

3. Downregulation of TGF-beta1 mRNA and protein in the muscles of patients with inflammatory myopathies after treatment with high-dose intravenous immunoglobulin;Amemiya;Clin. Immunol.,2000

4. Duchenne muscular dystrophy: deficiency of dystrophin at the muscle cell surface;Bonilla;Cell,1988

5. Contractile properties of skeletal muscles from young, adult and aged mice;Brooks;J. Physiol.,1988

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