Roles of NADPH oxidase and mitochondria in flow-induced vasodilation of human adipose arterioles: ROS-induced ROS release in coronary artery disease-Reference-Cited by-同舟云学术

Roles of NADPH oxidase and mitochondria in flow-induced vasodilation of human adipose arterioles: ROS-induced ROS release in coronary artery disease

Published:2017-08 Issue:6 Volume:24 Page:e12380
ISSN:1073-9688
Container-title:Microcirculation
language:en
Short-container-title:Microcirculation

Author:

Zinkevich Natalya S.¹²,Fancher Ibra S.³^ORCID,Gutterman David D.¹,Phillips Shane A.⁴

Affiliation:

1. Cardiovascular Center; Department of Medicine; Medical College of Wisconsin; Milwaukee WI USA

2. Department of Health and Medicine; Carroll University; Waukesha WI USA

3. Department of Physical Therapy; Department of Medicine (Division of Pulmonary, Critical Care, Sleep and Allergy); University of Illinois at Chicago; Chicago IL USA

4. Department of Physical Therapy; Department of Medicine (Division of Endocrinology, Diabetes and Metabolism); Integrative Physiology Laboratory; University of Illinois at Chicago; Chicago IL USA

Funder

National Heart, Lung, and Blood Institute

American Heart Association

Publisher

Wiley

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Molecular Biology,Physiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/micc.12380/fullpdf

Reference29 articles.

1. Corelease of nitric oxide and prostaglandins mediates flow- dependent dilation of rat gracilis muscle arterioles;Koller;Am J Physiol,1994

2. Enhanced release of prostaglandins contributes to flow-induced arteriolar dilation in eNOS knockout mice;Sun;Circ Res,1999