Role of moesin, Src, and ROS in advanced glycation end product-induced vascular endothelial dysfunction-Reference-Cited by-同舟云学术

Role of moesin, Src, and ROS in advanced glycation end product-induced vascular endothelial dysfunction

Published:2017-04 Issue:3 Volume:24 Page:e12358
ISSN:1073-9688
Container-title:Microcirculation
language:en
Short-container-title:Microcirculation

Author:

Zhang Wei-jin¹,Li Pei-xin¹,Guo Xiao-hua²,Huang Qiao-bing²^ORCID

Affiliation:

1. First Clinical College of Medicine; Southern Medical University; Guangzhou China

2. Department of Pathophysiology; Key Laboratory for Shock and Microcirculation Research of Guangdong Province; Southern Medical University; Guangzhou China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Molecular Biology,Physiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/micc.12358/fullpdf

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3. Advanced glycation end products and vascular inflammation: implications for accelerated atherosclerosis in diabetes;Basta;Cardiovasc Res,2004

4. Methylglyoxal promotes oxidative stress and endothelial dysfunction;Sena;Pharmacol Res,2012

5. The amino-terminal domains of the ezrin, radixin, and moesin (ERM) proteins bind advanced glycation end products, an interaction that may play a role in the development of diabetic complications;McRobert;J Biol Chem,2003

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