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Inhibition of proteasome-mediated glucocorticoid receptor degradation restores nitric oxide bioavailability in myocardial endothelial cellsin vitro

  • Published:2014-05-23 Issue:7 Volume:106 Page:219-235
  • ISSN:0248-4900
  • Container-title:Biology of the Cell
  • language:en
  • Short-container-title:Biol. Cell

Author:

Blecharz Kinga G.1,Burek Malgorzata1,Bauersachs Johann2,Thum Thomas3,Tsikas Dimitrios4,Widder Julian2,Roewer Norbert1,Förster Carola Y.1

Affiliation:

1. University of Würzburg; Department of Anaesthesia and Critical Care; Würzburg 97080 Germany

2. Hannover Medical School; Klinik für Kardiologie und Angiologie; Hannover 30625 Germany

3. Hannover Medical School; Institute for Molecular and Translational Therapeutic Strategies; Hannover 30625 Germany

4. Hannover Medical School; Department of Clinical Pharmacology; Hannover 30625 Germany

Publisher

Wiley

Subject

Cell Biology,General Medicine

Reference83 articles.

1. Glucocorticoid excess and hypertension;Baid;Curr. Hypertens. Rep.,2004

2. Induction of NO synthase in rat cardiac microvascular endothelial cells by IL-1 beta and IFN-gamma;Balligand;Am. J. Physiol.,1995

3. Gene regulation by steroid hormones;Beato;Cell,1989

4. Glucocorticoids increase VE-cadherin expression and cause cytoskeletal rearrangements in murine brain endothelial cEND cells;Blecharz;J. Cereb. Blood Flow Metab.,2008

5. Insights into glucocorticoid-associated hypertension;Brem;Am. J. Kidney Dis.,2001
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