Protein kinase Cε and protein kinase Cθ double‐deficient mice have a bleeding diathesis-Reference-Cited by-同舟云学术

Protein kinase Cε and protein kinase Cθ double‐deficient mice have a bleeding diathesis

Published:2012-09 Issue:9 Volume:10 Page:1887-1894
ISSN:1538-7933
Container-title:Journal of Thrombosis and Haemostasis
language:en
Short-container-title:Journal of Thrombosis and Haemostasis

Author:

UNSWORTH A. J.¹,FINNEY B. A.²,NAVARRO‐NUNEZ L.²,SEVERIN S.²,WATSON S. P.²,PEARS C. J.¹

Affiliation:

1. Department of Biochemistry, University of Oxford, Oxford

2. Centre for Cardiovascular Sciences, Institute of Biomedical Research, School of Clinical and Experimental Medicine, College of Medical and Dental Sciences, University of Birmingham, Birmingham, UK

Publisher

Wiley

Subject

Hematology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/j.1538-7836.2012.04857.x

Reference27 articles.

1. Synergy between Ca2+ and protein kinase C is the major factor in determining the level of secretion from human platelets

2. Protein kinase C- and calcium-regulated pathways independently synergize with Gi pathways in agonist-induced fibrinogen receptor activation

3. Molecular Mechanism of Thromboxane A2-induced Platelet Aggregation

4. Differential Role of Protein Kinase Cδ Isoform in Agonist-induced Dense Granule Secretion in Human Platelets

5. Regulation of Outside-in Signaling in Platelets by Integrin-associated Protein Kinase Cβ

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