Prevalence of the JAK2 V617F mutation is low among unselected patients with a first episode of unprovoked venous thromboembolism
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1538-7836.2007.02811.x/fullpdf
Reference19 articles.
1. A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera;James;Nature,2005
2. A gain-of-function mutation of JAK2 in myeloproliferative disorders;Kralovics;N Engl J Med,2005
3. Activating mutation in the tyrosine kinase JAK2 in polycythemia vera, essential thrombocythemia, and myeloid metaplasia with myelofibrosis;Levine;Cancer Cell,2005
4. Acquired mutation of the tyrosine kinase JAK2 in human myeloproliferative disorders;Baxter;Lancet,2005
5. The role of JAK2 V617F mutation, spontaneous erythropoiesis and megakaryocytopoiesis, hypersensitive platelets, activated leukocytes, and endothelial cells in the etiology of thrombotic manifestations in polycythemia vera and essential thrombocythemia;Bellucci;Semin Thromb Hemost,2006
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1. Deep vein thrombosis, pulmonary embolism, and portal vein thrombosis occurred in the same patient with the JAK2-V617F mutation treated with AngioJet-assisted thrombectomy via transjugular intrahepatic route;Journal of Vascular Surgery: Venous and Lymphatic Disorders;2023-05
2. Clinical epidemiology of venous thromboembolic disease: An institutional registry;Frontiers in Cardiovascular Medicine;2022-07-22
3. Quel bilan étiologique réaliser au décours d’une EP/TVP ?;Revue des Maladies Respiratoires;2021-04
4. JAK2V617F positive polycythemia vera with paroxysmal nocturnal hemoglobinuria and visceral thromboses: a case report and review of the literature;Thrombosis Journal;2021-03-10
5. JAK2V617F and calreticulin mutations in recurrent venous thromboembolism: results from the EDITH prospective cohort;Annals of Hematology;2016-10-20
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