Carboxypeptidase U (TAFIa) activity is induced in vivo in ischemic stroke patients receiving thrombolytic therapy
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1538-7836.2007.02798.x/fullpdf
Reference13 articles.
1. Established treatments for acute ischemic stroke;Khaja;Lancet,2007
2. Admission fibrinolytic profile is associated with symptomatic haemorrhagic transformation in stroke patients with tissue plasminogen activator;Ribo;Stroke,2004
3. The discovery of TAFI;Hendriks;J Thromb Haemost,2006
4. A study of the mechanism of inhibition of fibrinolysis by activated thrombin activable fibrinolysis inhibitor;Wang;J Biol Chem,1998
5. A role for procarboxypeptidase U in thrombosis;Willemse;Front Biosci,2007
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1. Structure/characteristics of thrombin activatable fibrinolysis inhibitor (TAFI) and its assay method;Japanese Journal of Thrombosis and Hemostasis;2023
2. Carboxypeptidase U (TAFIa) Is Rapidly Activated and Deactivated Following Thrombolysis and Thrombectomy in Stroke Patients;Translational Stroke Research;2021-11-19
3. Carboxypeptidase U (CPU, TAFIa, CPB2) in Thromboembolic Disease: What Do We Know Three Decades after Its Discovery?;International Journal of Molecular Sciences;2021-01-17
4. Inhibition of the procarboxypeptidase U (pro CPU , TAFI , pro CPB 2) system due to hemolysis;Journal of Thrombosis and Haemostasis;2019-05-02
5. Procarboxypeptidase U (proCPU, TAFI, proCPB2) in cerebrospinal fluid during ischemic stroke is associated with stroke progression, outcome and blood-brain barrier dysfunction;Journal of Thrombosis and Haemostasis;2017-12-26
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