Increase in plasma plasminogen activators inhibitor type 1 concentration after fibrinolytic treatment in patients with acute myocardial infarction is associated with 4G/5G polymorphism of PAI-1 gene
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1538-7836.2006.01990.x/fullpdf
Reference38 articles.
1. Environmental and Genetic Factors in Relation to Elevated Circulating Levels of Plasminogen Activator Inhibitor-1 in Caucasian Patients with Non-Insulin-Dependent Diabetes Mellitus
2. Genetic variation at the plasminogen activator inhibitor-1 locus is associated with altered levels of plasma plasminogen activator inhibitor-1 activity.
3. Serial Changes in Plasma Concentration of Plasminogen Activator Inhibitor-1 before and Serially after Thrombolytic Therapy for Acute Myocardial Infarction
4. Rebound increase of plasminogen activator inhibitor type 1 after cessation of thrombolytic treatment for acute myocardial infarction is independent of type of plasminogen activator used
5. Regulation of PAI-1 Expression by Genetic Polymorphisms
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1. Quantile‐specific heritability of plasminogen activator inhibitor type‐1 (PAI‐1, aka SERPINE1) and other hemostatic factors;Journal of Thrombosis and Haemostasis;2021-08-08
2. Role of Polymorphisms in Factor V (FV Leiden), Prothrombin, Plasminogen Activator Inhibitor Type-1 (PAI-1), Methylenetetrahydrofolate Reductase (MTHFR) and Cystathionine β-Synthase (CBS) Genes as Risk Factors for Thrombophilias;Mini-Reviews in Medicinal Chemistry;2012-09-01
3. Myocardial infarction and arterial thrombosis in identical newborn twins with homozygosity for the PAI-1 4 G/5 G polymorphism;International Journal of Cardiology;2009-09
4. Yin and yang of the plasminogen activator inhibitor;Polish Archives of Internal Medicine;2009-06-01
5. The influence of acute-phase levels of haemostatic factors on reperfusion and mortality in patients with acute myocardial infarction treated with streptokinase;Journal of Thrombosis and Thrombolysis;2007-08-25
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