Chromatin accessibility of kidney tubular cells under stress reveals key transcription factor mediating acute and chronic kidney disease-Reference-Cited by-同舟云学术

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Chromatin accessibility of kidney tubular cells under stress reveals key transcription factor mediating acute and chronic kidney disease

Published:2021-03-29 Issue:18 Volume:288 Page:5446-5458
ISSN:1742-464X
Container-title:The FEBS Journal
language:en
Short-container-title:FEBS J

Author:

Xing Yuexian¹²,Wang Qi²,Zhang Jing²,Li Wenju²,Duan Aiping²,Yang Jingping²³^ORCID,Liu Zhihong¹²

Affiliation:

1. National Clinical Research Center of Kidney Diseases Jinling Hospital School of Medicine Southeast University Nanjing China

2. National Clinical Research Center of Kidney Diseases Jinling Hospital Medical School Nanjing University Nanjing China

3. Jiangsu Key Laboratory of Molecular Medicine Medical School Nanjing University Nanjing China

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/febs.15818

Reference47 articles.

1. Renal tubule injury: a driving force toward chronic kidney disease

2. A new Twist in kidney fibrosis

3. Gene expression databases for kidney epithelial cells

4. Long-range enhancer–promoter contacts in gene expression control

5. Gene Regulatory Elements, Major Drivers of Human Disease

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1. The activity of early-life gene regulatory elements is hijacked in aging through pervasive AP-1-linked chromatin opening;Cell Metabolism;2024-08

2. The chromatin remodeling protein BRG1 mediates Ang II induced pro-fibrogenic response in renal fibroblasts;Life Sciences;2024-03

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