LGI1 downregulation increases neuronal circuit excitability-Reference-Cited by-同舟云学术

登录注册会员服务联系我们

LGI1 downregulation increases neuronal circuit excitability

Published:2020-10-26 Issue:12 Volume:61 Page:2836-2846
ISSN:0013-9580
Container-title:Epilepsia
language:en
Short-container-title:Epilepsia

Author:

Lugarà Eleonora¹^ORCID,Kaushik Rahul²³,Leite Marco¹,Chabrol Elodie¹,Dityatev Alexander²³⁴,Lignani Gabriele¹^ORCID,Walker Matthew C.¹

Affiliation:

1. Department of Clinical and Experimental Epilepsy UCL Queen Square Institute of Neurology London UK

2. German Center for Neurodegenerative Diseases Magdeburg Germany

3. Center for Behavioral Brain Sciences Magdeburg Germany

4. Medical Faculty Otto von Guericke University Magdeburg Germany

Funder

H2020 Marie Skłodowska-Curie Actions

Epilepsy Research UK

Publisher

Wiley

Subject

Clinical Neurology,Neurology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/epi.16736

Reference46 articles.

1. Disruption of LGI1–linked synaptic complex causes abnormal synaptic transmission and epilepsy

2. A novel gene, LGI1, from 10q24 is rearranged and downregulated in malignant brain tumors

3. Auditory features;Kalachikov S;Am J Hum Genet,2002

4. Mutations in the LGI1/Epitempin gene on 10q24 cause autosomal dominant lateral temporal epilepsy

5. Electroclinical characterization of epileptic seizures in leucine-rich, glioma-inactivated 1-deficient mice

Cited by 16 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. OPALIN is an LGI1 receptor promoting oligodendrocyte differentiation;Proceedings of the National Academy of Sciences;2024-07-31

2. Activity-driven synaptic translocation of LGI1 controls excitatory neurotransmission;Cell Reports;2024-05

3. Peripherally-derived LGI1-reactive monoclonal antibodies cause epileptic seizures in vivo;Brain;2024-04-25

4. Molecular dissection of an immunodominant epitope in Kv1.2-exclusive autoimmunity;Frontiers in Immunology;2024-04-11

5. Pathophysiological Effects of Autoantibodies in Autoimmune Encephalitides;Cells;2023-12-20

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号京ICP备18003416号-3