Inhibition of receptor‐interacting protein kinase‐3 in the necroptosis pathway attenuates inflammatory bone loss in experimental apical periodontitis in Balb/c mice
Author:
Affiliation:
1. State Key Laboratory of Oral Diseases & National Clinical Research Center for Oral Diseases Department of Prosthodontics West China Hospital of Stomatology Sichuan University Sichuan China
Funder
National Natural Science Foundation of China
Publisher
Wiley
Subject
General Dentistry
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/iej.13534
Reference29 articles.
1. Fusobacterium nucleatum — symbiont, opportunist and oncobacterium
2. Enterococcus faecalis induces necroptosis in human osteoblastic MG63 cells through the RIPK3 / MLKL signalling pathway
3. RIPK1 maintains epithelial homeostasis by inhibiting apoptosis and necroptosis
4. Effect of intercellular adhesion molecule 1 deficiency on the development of apical periodontitis
5. Apical Periodontitis Is Associated with Elevated Concentrations of Inflammatory Mediators in Peripheral Blood: A Systematic Review and Meta-analysis
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2. Necroptosis in apical periodontitis: A programmed cell death with multiple roles;Journal of Cellular Physiology;2023-07-11
3. A novel experimental model to investigate fungal involvement shows expression of Dectin‐1 in periapical lesion pathogenesis;Journal of Oral Rehabilitation;2023-06-09
4. RGS10 negatively regulates apical periodontitis via TFEB‐mediated autophagy in BABL/c mice model and in vitro;International Endodontic Journal;2023-05-06
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