Secondary fusion proteins as a mechanism of <scp>BCR</scp> :: <scp>ABL1</scp> kinase‐independent resistance in chronic myeloid leukaemia-Reference-Cited by-同舟云学术

Secondary fusion proteins as a mechanism of BCR :: ABL1 kinase‐independent resistance in chronic myeloid leukaemia

Published:2022-10-20 Issue:3 Volume:200 Page:323-328
ISSN:0007-1048
Container-title:British Journal of Haematology
language:en
Short-container-title:Br J Haematol

Author:

Barnes Evan J.¹^ORCID,Eide Christopher A.¹,Kaempf Andy²,Bottomly Daniel³,Romine Kyle A.¹,Wilmot Beth³,Saunders Dominick⁴,McWeeney Shannon K.³,Tognon Cristina E.¹^ORCID,Druker Brian J.¹

Affiliation:

1. Division of Hematology and Medical Oncology Knight Cancer Institute, Oregon Health & Science University Portland Oregon USA

2. Biostatistics Shared Resource Knight Cancer Institute, Oregon Health & Science University Portland Oregon USA

3. Division of Bioinformatics and Computational Biology, Department of Medical Informatics and Clinical Epidemiology Knight Cancer Institute, Oregon Health & Science University Portland Oregon USA

4. Flow Cytometry Shared Resource Knight Cancer Institute, Oregon Health & Science University Portland Oregon USA

Funder

American Society of Hematology

National Cancer Institute

Publisher

Wiley

Subject

Hematology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/bjh.18515

Reference15 articles.

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