Three Japanese adult cases of brain lesions with anti-myelin oligodendrocyte glycoprotein antibodies lacking optic neuritis and myelitis

Author:

Sawada Jun1,Katayama Takayuki1,Toyoshima Sarasa1,Nitamizu Senri1,Yamamoto Kenji1,Fukuura Ai1,Kikuchi Shiori1,Kano Kohei1,Asanome Asuka1,Takahashi Kae1,Saito Tsukasa1,Takahashi Toshiyuki23,Kaneko Kimihiko3,Nakashima Ichiro4,Hasebe Naoyuki1

Affiliation:

1. Division of Neurology; Department of Internal Medicine; Asahikawa Medical University; Asahikawa Hokkaido Japan

2. Department of Neurology; Yonezawa National Hospital; Yonezawa Yamagata Japan

3. Department of Neurology; Tohoku University School of Medicine; Sendai Miyagi Japan

4. Department of Neurology; Tohoku Medical and Pharmaceutical University School of Medicine; Sendai Miyagi Japan

Funder

Mitsubishi Tanabe Pharma Corporation

Life Sciences Institute

Publisher

Wiley

Subject

Clinical Neurology,Immunology and Microbiology (miscellaneous),Immunology,Neuroscience (miscellaneous)

Reference20 articles.

1. Anti-aquaporin-4 antibody is involved in the pathogenesis of NMO: a study on antibody titre;Takahashi;Brain,2007

2. Distinction between MOG antibody-positive and AQP4 antibody-positive NMO spectrum disorders;Sato;Neurology,2014

3. International consensus diagnostic criteria for neuromyelitis optica spectrum disorders;Wingerchuk;Neurology,2015

4. MOG-IgG in NMO and related disorders: a multicenter study of 50 patients. Part 1: frequency, syndrome specificity, influence of disease activity, long-term course, association with AQP4-IgG, and origin;Jarius;J Neuroinflammation,2016a

5. The clinical spectrum associated with myelin oligodendrocyte glycoprotein antibodies (anti-MOG-Ab) in Thai patients;Siritho;Mult Scler,2016

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