Interferon β-1a reduces increased interleukin-16 levels in multiple sclerosis patients

Author:

Nischwitz S.1,Faber H.1,Sämann P. G.2,Domingues H. S.34,Krishnamoorthy G.3,Knop M.1,Müller-Sarnowski F.1,Yassouridis A.5,Weber F.1

Affiliation:

1. RG Inflammatory Disorders of the CNS, Neurology; Max Planck Institute of Psychiatry; Munich Germany

2. RG Neuroimaging; Max Planck Institute of Psychiatry; Munich Germany

3. Max Planck Institute of Neurobiology; Martinsried Germany

4. Instituto de Biologia Molecular e Celular R. Campo Alegre; Porto Portugal

5. RG Biostatistics; Max Planck Institute of Psychiatry; Munich Germany

Publisher

Wiley

Subject

Neurology (clinical),Neurology,General Medicine

Reference29 articles.

1. Multiple sclerosis;Compston;Lancet,2008

2. Staging of multiple sclerosis (MS) lesions: pathology of the time frame of MS;Valk;Neuropathol Appl Neurobiol,2000

3. Interleukin- 16;Cruikshank;J Leukoc Biol,2000

4. Processing and release of IL-16 from CD4+ but not CD8+ T-cells is activation dependent;Wu;J Immunol,1999

5. Not-so-sweet sixteen: the role of IL-16 in infectious and immune-mediated inflammatory diseases;Glass;J Interferon Cytokine Res,2006

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