Deficient glucose uptake is linked to impaired Glut1 expression upon CD3/CD28 stimulation in memory T cells from pleural effusions secondary to lung cancer-Reference-Cited by-同舟云学术

Deficient glucose uptake is linked to impaired Glut1 expression upon CD3/CD28 stimulation in memory T cells from pleural effusions secondary to lung cancer

Published:2019-07-21 Issue:3 Volume:90 Page:
ISSN:0300-9475
Container-title:Scandinavian Journal of Immunology
language:en
Short-container-title:Scand J Immunol

Author:

Prado‐Garcia Heriberto¹^ORCID,Romero‐Garcia Susana¹^ORCID,Castro‐Flores Daniela Alejandra¹,Rumbo‐Nava Uriel²

Affiliation:

1. Departamento de Enfermedades Cronico‐Degenerativas Instituto Nacional de Enfermedades Respiratorias, “Ismael Cosio Villegas” Mexico City Mexico

2. Clinica de Neumo‐Oncologia Instituto Nacional de Enfermedades Respiratorias, “Ismael Cosio Villegas” Mexico City Mexico

Funder

Consejo Nacional de Innovación, Ciencia y Tecnología

Publisher

Wiley

Subject

Immunology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/sji.12802

Reference29 articles.

1. Cell surface Glut1 levels distinguish human CD4 and CD8 T lymphocyte subsets with distinct effector functions

2. Immunometabolic Regulations Mediated by Coinhibitory Receptors and Their Impact on T Cell Immune Responses

3. Glucose Uptake Is Limiting in T Cell Activation and Requires CD28-Mediated Akt-Dependent and Independent Pathways

4. The Glucose Transporter Glut1 Is Selectively Essential for CD4 T Cell Activation and Effector Function

5. Anergic T Cells Are Metabolically Anergic

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