Aging‐induced aberrant RAGE/PPARα axis promotes hepatic steatosis via dysfunctional mitochondrial β oxidation-Reference-Cited by-全球学者库

Aging‐induced aberrant RAGE/PPARα axis promotes hepatic steatosis via dysfunctional mitochondrial β oxidation

Published:2020-09-16 Issue:10 Volume:19 Page:
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Wan Jian¹,Wu Xiangsong²,Chen Hanbei³,Xia Xinyi⁴,Song Xi¹,Chen Song¹,Lu Xinyuan¹,Jin Jie³,Su Qing³,Cai Dongsheng⁵^ORCID,Liu Bin⁶,Li Bo³^ORCID

Affiliation:

1. Department of Emergency and Critical Care Medicine Shanghai Pudong New Area People's Hospital Shanghai University of Medicine and Health Sciences Shanghai China

2. Department of General Surgery XinHua Hospital Shanghai Jiao Tong University School of Medicine Shanghai China

3. Department of Endocrinology XinHua Hospital Shanghai Jiao Tong University School of Medicine Shanghai China

4. Department of Endocrinology and Metabolism Shanghai Jiao Tong University Affiliated Sixth People's Hospital Shanghai China

5. Department of Molecular Pharmacology Diabetes Research Center Institute of Aging Albert Einstein College of Medicine Bronx NY USA

6. Hubei Key Laboratory for Kidney Disease Pathogenesis and Intervention Hubei Polytechnic University School of Medicine Huangshi China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Aging

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.13238

Reference56 articles.

1. The acidic tail of HMGB1 regulates its secondary structure and conformational flexibility: A circular dichroism and molecular dynamics simulation study

2. Critical role of RAGE and HMGB1 in inflammatory heart disease

3. Diabetes Increases Formation of Advanced Glycation End Products on Sarco(endo)plasmic Reticulum Ca2+-ATPase

4. Multiple levels of regulation determine the role of the receptor for AGE (RAGE) as common soil in inflammation, immune responses and diabetes mellitus and its complications

5. RAGE deficiency does not affect non-alcoholic steatohepatitis and atherosclerosis in Western type diet-fed Ldlr−/− mice

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