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Preserving transcriptional stress responses as an anti‐aging strategy

Published:2021-01-20 Issue:2 Volume:20 Page:
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Cheng Yang¹^ORCID,Pitoniak Andrew¹^ORCID,Wang Julia²^ORCID,Bohmann Dirk¹^ORCID

Affiliation:

1. Department of Biomedical Genetics University of Rochester Medical Center Rochester New York USA

2. Medical Scientist Training Program Baylor College of Medicine Houston Texas USA

Funder

Glenn Foundation for Medical Research

National Institute of Dental and Craniofacial Research

National Institute on Aging

Publisher

Wiley

Subject

Cell Biology,Aging

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.13297

Reference48 articles.

1. Genome‐wide dFOXO targets and topology of the transcriptomic response to stress and insulin signalling

2. Epigenetic Involvement in Hutchinson-Gilford Progeria Syndrome: A Mini-Review

3. Neuronal Expression of p53 Dominant-Negative Proteins in Adult Drosophila melanogaster Extends Life Span

4. Determinants and dynamics of genome accessibility

5. Collapse of proteostasis represents an early molecular event in Caenorhabditis elegans aging

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2. Activation of the Keap1/Nrf2 pathway suppresses mitochondrial dysfunction, oxidative stress, and motor phenotypes inC9orf72ALS/FTD models;Life Science Alliance;2024-06-21

3. Thioredoxin (Trx): A redox target and modulator of cellular senescence and aging-related diseases;Redox Biology;2024-04

4. A PI3K-calcium-Nox axis primes leukocyte Nrf2 to boost immune resilience and limit collateral damage;Journal of Cell Biology;2023-03-30

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