PLA2G16 is a mutant p53/KLF5 transcriptional target and promotes glycolysis of pancreatic cancer-Reference-Cited by-同舟云学术

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PLA2G16 is a mutant p53/KLF5 transcriptional target and promotes glycolysis of pancreatic cancer

Published:2020-09-27 Issue:21 Volume:24 Page:12642-12655
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J. Cell. Mol. Med.

Author:

Xia Wei¹^ORCID,Bai Hansong²,Deng Ying²,Yang Yi¹

Affiliation:

1. Department of Endocrinology Sichuan Provincial People's Hospital University of Electronic Science and Technology of China Chengdu China

2. Cancer Center Sichuan Provincial People's Hospital University of Electronic Science and Technology of China Chengdu China

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.15832

Reference45 articles.

1. Cancer statistics, 2019

2. LAT2 regulates glutamine-dependent mTOR activation to promote glycolysis and chemoresistance in pancreatic cancer

3. Role of abnormal lipid metabolism in development, progression, diagnosis and therapy of pancreatic cancer

4. Transcriptional and translational downregulation of H-REV107, a class II tumour suppressor gene located on human chromosome 11q11-12

5. The class II tumour suppressor gene H-REV107-1 is a target of interferon-regulatory factor-1 and is involved in IFNγ-induced cell death in human ovarian carcinoma cells

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3. Britannin inhibits cell proliferation, migration and glycolysis by downregulating KLF5 in lung cancer;Experimental and Therapeutic Medicine;2024-01-22

4. Comparative transcriptome and metabolome analyses of wild and mutant Oujiang color common carp through editing SCARB1 gene by CRISPR/Cas technology;Aquaculture;2023-12

5. Abrogation of KLF5 sensitizes BRCA1-proficient pancreatic cancer to PARP inhibition;Acta Biochimica et Biophysica Sinica;2023-12-01

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