Vein graft disease in a knockout mouse model of hyperhomocysteinaemia

Author:

Steger Christina Maria1,Mayr Tobias2,Bonaros Nikolaos3,Bonatti Johannes4,Schachner Thomas3

Affiliation:

1. Department of Pathology; Academic Teaching Hospital Feldkirch (Affiliation of the Innsbruck Medical University); Feldkirch Austria

2. Department of Surgery; State Hospital Kufstein; Kufstein Austria

3. Department of Cardiac Surgery; Innsbruck Medical University; Innsbruck Austria

4. Heart and Vascular Institute; Cleveland Clinic Abu Dhabi; Abu Dhabi UAE

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Pathology and Forensic Medicine

Reference51 articles.

1. Vascular complications of cystathionine β-synthase deficiency: future directions for homocysteine-to-hydrogen sulfide research;Beard;Am. J. Physiol. Heart Circ. Physiol.,2011

2. Hyperhomocyst(e)inemia impairs angiogenesis in a murine model of limb ischemia;Bosch-Marcé;Vasc. Med.,2005

3. The formation of a homologue of cystine by the decomposition of methionine with sulfuric acid;Butz;J. Biol. Chem.,1932

4. Folic acid inhibits homocysteine-induced proliferation of human arterial smooth muscle cells;Carmody;J. Vasc. Surg.,1999

5. Metabolic abnormalities detected in a survey of mentally backward individuals in Northern Ireland;Carson;Arch. Dis. Child.,1962

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