Serum and blister‐fluid elevation and decreased epidermal content of high‐mobility group box 1 protein in drug‐induced Stevens–Johnson syndrome/toxic epidermal necrolysis-Reference-Cited by-同舟云学术

Serum and blister‐fluid elevation and decreased epidermal content of high‐mobility group box 1 protein in drug‐induced Stevens–Johnson syndrome/toxic epidermal necrolysis

Published:2019-03-26 Issue:1 Volume:181 Page:166-174
ISSN:0007-0963
Container-title:British Journal of Dermatology
language:en
Short-container-title:Br J Dermatol

Author:

Carr D.F.¹^ORCID,Wang C.‐W.²³,Bellón T.⁴,Ressel L.⁵,Nwikue G.¹,Shrivastava V.⁶,Bergfeld W.⁷,Jorgensen A.L.⁸,Chung W.‐H.²,Pirmohamed M.¹

Affiliation:

1. Department of Molecular and Clinical Pharmacology University of Liverpool Liverpool U.K.

2. Department of Dermatology Drug Hypersensitivity Clinical and Research Center Chang Gung Memorial Hospital Keelung, Linkou, Taipei Taiwan

3. College of Medicine Chang Gung University Taoyuan Taiwan

4. La Paz University Hospital Health Research Institute (IdiPAZ) Madrid Spain

5. Departments of Veterinary Pathology and Public Health University of Liverpool Liverpool U.K.

6. Naval Medical Center Portsmouth Portsmouth VA U.S.A.

7. Department of Dermatology and Dermatopathology Cleveland Clinic Foundation Cleveland OH U.S.A.

8. Departments of Biostatistics University of Liverpool Liverpool U.K.

Funder

Medical Research Council

Publisher

Wiley

Subject

Dermatology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/bjd.17610

Reference27 articles.

1. The Epidemiology of Stevens-Johnson Syndrome and Toxic Epidermal Necrolysis in the UK

2. DRG 272: Does it Provide Adequate Burn Center Reimbursement for the Care of Patients With Stevens–Johnson Syndrome and Toxic Epidermal Necrolysis?

3. Human leukocyte antigen and idiosyncratic adverse drug reactions

4. Granulysin is a key mediator for disseminated keratinocyte death in Stevens-Johnson syndrome and toxic epidermal necrolysis

5. Delayed reactions to drugs show levels of perforin, granzyme B, and Fas-L to be related to disease severity

Cited by 17 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. HMGB1: A molecular paradox linking skin injury, inflammation and repair;Indian Journal of Dermatology, Venereology and Leprology;2024-06-21

2. Greater mechanistic understanding of the cutaneous pathogenesis of Stevens–Johnson syndrome/toxic epidermal necrolysis can shed light on novel therapeutic strategies: a comprehensive review;Current Opinion in Allergy & Clinical Immunology;2024-05-17

3. Correlations between histopathologic findings, serum biomarker levels, and clinical outcomes in Stevens–Johnson syndrome/toxic epidermal necrolysis (SJS/TEN);Scientific Reports;2023-08-21

4. TNF‐α induced extracellular release of keratinocyte high‐mobility group box 1 in Stevens‐Johnson syndrome/toxic epidermal necrolysis: Biomarker and putative mechanism of pathogenesis;The Journal of Dermatology;2023-06-02

5. Dasatinib causes keratinocyte apoptosis via inhibiting high mobility group Box 1-mediated mitophagy;Toxicology Letters;2023-01