Clinical utility of complement-dependent C3d assay in kidney recipients presenting with late allograft dysfunction
Author:
Affiliation:
1. Department of Medicine; Division of Nephrology; University of British Columbia; Vancouver BC Canada
2. Department of Pathology and Laboratory Medicine; UCLA Immunogenetics Center; University of California, Los Angeles; Los Angeles CA USA
Funder
University of British Columbia
National Institutes of Health
Publisher
Wiley
Subject
Pharmacology (medical),Transplantation,Immunology and Allergy
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/ajt.14871/fullpdf
Reference31 articles.
1. Evidence for antibody-mediated injury as a major determinant of late kidney allograft failure;Gaston;Transplantation,2010
2. Novel C1q assay reveals a clinically relevant subset of human leukocyte antigen antibodies independent of immunoglobulin G strength on single antigen beads;Chen;Hum Immunol,2011
3. Clinical usefulness of a novel C1q assay to detect immunoglobulin G antibodies capable of fixing complement in sensitized pediatric heart transplant patients;Chin;J Heart Lung Transplant,2011
4. Complement-binding anti-HLA antibodies and kidney-allograft survival;Loupy;N Engl J Med,2013
5. C4d fixing, luminex binding antibodies-a new tool for prediction of graft failure after heart transplantation;Smith;Am J Transplant,2007
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2. Complement-activating donor-specific anti-HLA antibodies in solid organ transplantation: systematic review, meta-analysis, and critical appraisal;Frontiers in Immunology;2023-10-02
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