Myocardial infarction is sufficient to increase GLP-1 secretion, leading to improved left ventricular contractility and mitochondrial respiratory capacity
Author:
Affiliation:
1. Department of Internal Medicine I; University Hospital Aachen, RWTH Aachen University; Aachen Germany
2. Department of Translational Science; Comprehensive Heart Failure Center, University of Würzburg; Würzburg Germany
Funder
Hans Lamers foundation
Corona foundation
German Research Foundation (DFG) SFB/Transregio 219 (SFB-TRR) “Mechanisms of Cardiovascular Complications in Chronic Kidney Disease”
Boehringer Ingelheim
Publisher
Wiley
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism,Internal Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/dom.13472/fullpdf
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3. Cardiovascular actions and clinical outcomes with glucagon-like Peptide-1 receptor agonists and dipeptidyl Peptidase-4 inhibitors;Nauck;Circulation,2017
4. Cardioprotective and vasodilatory actions of glucagon-like peptide 1 receptor are mediated through both glucagon-like peptide 1 receptor-dependent and -independent pathways;Ban;Circulation,2008
5. The cardiovascular biology of glucagon-like Peptide-1;Drucker;Cell Metab,2016
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