Pre-synaptic C-terminal truncated tau is released from cortical synapses in Alzheimer's disease-Reference-Cited by-同舟云学术

Pre-synaptic C-terminal truncated tau is released from cortical synapses in Alzheimer's disease

Published:2015-01-13 Issue:3 Volume:133 Page:368-379
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Sokolow Sophie¹²³⁴,Henkins Kristen M.¹,Bilousova Tina¹,Gonzalez Bianca¹,Vinters Harry V.⁵⁶,Miller Carol A.⁷,Cornwell Lindsey⁸,Poon Wayne W.⁸,Gylys Karen H.¹²³⁶

Affiliation:

1. UCLA School of Nursing; Los Angeles California USA

2. UCLA Brain Research Institute; Los Angeles California USA

3. UCLA Center for the Advancement of Gerontological Nursing Sciences; Los Angeles California USA

4. UCLA Clinical and Translational Science Institute; Los Angeles California USA

5. Departments of Pathology and Laboratory Medicine; David Geffen School of Medicine at UCLA; Los Angeles California USA

6. Mary S. Easton Center for Alzheimer's Research at UCLA; Los Angeles California USA

7. Departments of Pathology; Neurology; and Program in Neuroscience; Keck USC School of Medicine; Los Angeles California USA

8. Institute for Memory Impairments and Neurological Disorders; UC Irvine; Irvine California USA

Funder

Alzheimer's association

UCLA School of Nursing

NIA

Daljit S. and Elaine Sarkaria Chair in Diagnostic Medicine

NIH

JCCC

UCLA AIDS Institute

David Geffen School of Medicine

Chancellor's Office at UCLA

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.12991/fullpdf

Reference74 articles.

1. A NH2 tau fragment targets neuronal mitochondria at AD synapses: possible implications for neurodegeneration;Amadoro;J. Alzheimers Dis.,2010

2. Interaction between NH(2)-tau fragment and Abeta in Alzheimer's disease mitochondria contributes to the synaptic deterioration;Amadoro;Neurobiol. Aging,2012

3. AD-linked, toxic NH2 human tau affects the quality control of mitochondria in neurons;Amadoro;Neurobiol. Dis.,2014a

4. Cerebrospinal fluid levels of a 20-22 kDa NH2 fragment of human tau provide a novel neuronal injury biomarker in Alzheimer's disease and other dementias;Amadoro;J. Alzheimers Dis.,2014b

5. Apolipoprotein E4 causes age- and Tau-dependent impairment of GABAergic interneurons, leading to learning and memory deficits in mice;Andrews-Zwilling;J. Neurosci.,2010

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