Molecular Correlates of Topiramate and GRIK1 rs2832407 Genotype in Pluripotent Stem Cell–Derived Neural Cultures-Reference-Cited by-同舟云学术

Molecular Correlates of Topiramate and GRIK1 rs2832407 Genotype in Pluripotent Stem Cell–Derived Neural Cultures

Published:2020-08 Issue:8 Volume:44 Page:1561-1570
ISSN:0145-6008
Container-title:Alcoholism: Clinical and Experimental Research
language:en
Short-container-title:Alcohol Clin Exp Res

Author:

Lieberman Richard¹²,Jensen Kevin P.³⁴,Clinton Kaitlin¹,Levine Eric S.²,Kranzler Henry R.⁵⁶,Covault Jonathan¹^ORCID

Affiliation:

1. From the Alcohol Research Center (RL, KC, JC) Department of Psychiatry University of Connecticut School of Medicine Farmington Connecticut USA

2. Department of Neuroscience (RL, ESL) University of Connecticut School of Medicine Farmington Connecticut USA

3. Department of Psychiatry (KPJ) Yale University School of Medicine New Haven Connecticut USA

4. VA Connecticut Healthcare System(KPJ) West Haven Connecticut USA

5. Center for Studies of Addiction (HRK) Department of Psychiatry Perelman School of Medicine of the University of Pennsylvania Philadelphia Pennsylvania USA

6. VISN4 MIRECC (HRK) Crescenz VAMC Philadelphia Pennsylvania USA

Funder

National Institute on Alcohol Abuse and Alcoholism

Publisher

Wiley

Subject

Psychiatry and Mental health,Toxicology,Medicine (miscellaneous)

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acer.14399

Reference54 articles.

1. Topiramate modulation of kainate-induced calcium currents is inversely related to channel phosphorylation level

2. Presynaptic facilitation of glutamate release in the basolateral amygdala: A mechanism for the anxiogenic and seizurogenic function of GluK1 receptors

3. Comparing topiramate with naltrexone in the treatment of alcohol dependence*

4. Genomic organization, proposed alternative splicing mechanisms, and RNA editing structure of GRIK1

5. Topiramate Reduces Excitability in the Basolateral Amygdala by Selectively Inhibiting GluK1 (GluR5) Kainate Receptors on Interneurons and Positively Modulating GABAA Receptors on Principal Neurons

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