Involvement of ICAM-1 in impaired spermatogenesis after busulfan treatment in mice-Reference-Cited by-同舟云学术

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Involvement of ICAM-1 in impaired spermatogenesis after busulfan treatment in mice

Published:2015-03-25 Issue:1 Volume:48 Page:37-44
ISSN:0303-4569
Container-title:Andrologia
language:en
Short-container-title:Andrologia

Author:

Cai Y.¹,Liu T.²,Fang F.¹,Shen S.³,Xiong C.¹⁴

Affiliation:

1. Family Planning Research Institute; Tongji Medical College; Huazhong University of Science and Technology; Wuhan China

2. Department of Thoracic Surgery; Renmin Hospital of Wuhan University; Wuhan China

3. Zhong Shen Bioscience Inc.; Wuhan China

4. Center for Reproductive Medicine; Tongji Medical College; Huazhong University of Science and Technology; Wuhan China

Funder

National Science & Technology Pillar Program during the 12th 5-Year Plan Period, China

Independent innovation of Huazhong University of Science and Technology, China

Wuhan High-tech District

Publisher

Wiley

Subject

Urology,Endocrinology,General Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/and.12414/fullpdf

Reference23 articles.

1. Chronic cyclophosphamide treatment alters the expression of stress response genes in rat male germ cells;Aguilar-Mahecha;Biol Reprod,2002

2. The cycle of the seminiferous epithelium in humans: a need to revisit?;Amann;J Androl,2008

3. Effects of busulfan on murine spermatogenesis: cytotoxicity, sterility, sperm abnormalities, and dominant lethal mutations;Bucci;Mutat Res,1987

4. The blood-testis barrier and its implications for male contraception;Chen;Pharmacol Rev,2012

5. Effects of multiple doses of cyclophosphamide on mouse testes: accessing the germ cells lost, and the functional damage of stem cells;Drumond;Reprod Toxicol,2011

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1. Effect of platelet rich plasma versus melatonin on testicular injury induced by Busulfan in adult albino rats: a histological and immunohistochemical study;Ultrastructural Pathology;2024-02-29

2. LPS-Induced Systemic Inflammation Caused mPOA-FSH/LH Disturbance and Impaired Testicular Function;Frontiers in Endocrinology;2022-06-23

3. Endoplasmic reticulum stress promotes blood-testis barrier impairment in mice with busulfan-induced oligospermia through PERK-eIF2α signaling pathway;Toxicology;2022-05

4. Identification of a Crosstalk among TGR5, GLIS2, and TP53 Signaling Pathways in the Control of Undifferentiated Germ Cell Homeostasis and Chemoresistance;Advanced Science;2022-04-18

5. Busulfan impairs blood–testis barrier and spermatogenesis by increasing noncollagenous 1 domain peptide via matrix metalloproteinase 9;Andrology;2021-09-30

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