Phenobarbital, midazolam, bumetanide, and neonatal seizures: The devil is in the details
Author:
Affiliation:
1. Neurochlore, Fundamental Research Department Ben‐Ari Institute of Neuroarcheology Marseille France
2. Department of Anesthesiology Vanderbilt University Medical School Nashville Tennessee USA
Funder
National Institutes of Health
Publisher
Wiley
Subject
Clinical Neurology,Neurology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/epi.16830
Reference53 articles.
1. Lost in translation: the valley of death across preclinical and clinical divide – identification of problems and overcoming obstacles
2. Phenobarbital and midazolam suppress neonatal seizures in a noninvasive rat model of birth asphyxia, whereas bumetanide is ineffective
3. NKCC1 transporter facilitates seizures in the developing brain
4. Bumetanide enhances phenobarbital efficacy in a neonatal seizure model
5. Neuronal chloride accumulation and excitatory GABA underlie aggravation of neonatal epileptiform activities by phenobarbital
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