Loss of Ethanol Inhibition of N -Methyl-D-Aspartate Receptor-Mediated Currents and Plasticity of Cerebellar Synapses in Mice Expressing the GluN1(F639A) Subunit-Reference-Cited by-同舟云学术

Loss of Ethanol Inhibition of N -Methyl-D-Aspartate Receptor-Mediated Currents and Plasticity of Cerebellar Synapses in Mice Expressing the GluN1(F639A) Subunit

Published:2018-02-09 Issue:4 Volume:42 Page:698-705
ISSN:0145-6008
Container-title:Alcoholism: Clinical and Experimental Research
language:en
Short-container-title:Alcohol Clin Exp Res

Author:

Zamudio-Bulcock Paula A.¹,Homanics Gregg E.²,Woodward John J.¹^ORCID

Affiliation:

1. Department of Neuroscience; Medical University of South Carolina; Charleston South Carolina

2. Department of Anesthesiology; Univeristy of Pittsburgh; Pittsburgh PA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Psychiatry and Mental health,Toxicology,Medicine (miscellaneous)

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/acer.13597/fullpdf

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4. Ethanol sensitivity of GABAergic currents in cerebellar granule neurons is not increased by a single amino acid change (R100Q) in the alpha6 GABAA receptor subunit;Botta;J Pharmacol Exp Ther,2007

5. Burst-dependent bidirectional plasticity in the cerebellum is driven by presynaptic NMDA receptors;Bouvier;Cell Rep,2016

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