Sulodexide attenuates endoplasmic reticulum stress induced by myocardial ischaemia/reperfusion by activating the PI3K/Akt pathway-Reference-Cited by-同舟云学术

Sulodexide attenuates endoplasmic reticulum stress induced by myocardial ischaemia/reperfusion by activating the PI3K/Akt pathway

Published:2019-05-23 Issue:8 Volume:23 Page:5063-5075
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J Cell Mol Med

Author:

Shen Danping¹²,Chen Ruiyao¹,Zhang Lijing¹,Rao Zhiheng¹,Ruan Yongxue¹,Li Lei¹,Chu Maoping¹^ORCID,Zhang Yuanhai¹

Affiliation:

1. Children's Heart Center Institute of Cardiovascular Development and Translational Medicine, The Second Affiliated Hospital & Yuying Children's Hospital of Wenzhou Medical University Wenzhou Zhejiang China

2. The Children's Hospital Zhejiang University School of Medicine Hangzhou Zhejiang China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.14367

Reference42 articles.

1. Clinical manifestations and basic mechanisms of myocardial ischemia/reperfusion injury;Yang CF;Ci Ji Yi Xue Za Zhi,2018

2. Assessing the Global Burden of Ischemic Heart Disease: Part 2: Analytic Methods and Estimates of the Global Epidemiology of Ischemic Heart Disease in 2010

3. Reperfusion injury in acute myocardial infarction. From bench to cath lab. Part I: Basic considerations

4. Diazoxide postconditioning induces mitochondrial protein S-Nitrosylation and a redox-sensitive mitochondrial phosphorylation/translocation of RISK elements: no role for SAFE

5. Ischemic postconditioning and pinacidil suppress calcium overload in anoxia-reoxygenation cardiomyocytes via down-regulation of the calcium-sensing receptor

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