Fatty acid synthase and Ki‐67 immunoexpression can be useful for the identification of malignant component in carcinoma ex‐pleomorphic adenoma

Author:

Díaz Katya Pulido1,Gondak Rogério2ORCID,Martins Leandro Liporoni3,Almeida Oslei Paes4,León Jorge Esquiche5,Mariano Fernanda Viviane6,Altemani Albina6,Vargas Pablo Agustin4

Affiliation:

1. Universidad Autónoma de Baja California, Escuela de Ciencias de la Salud (UABC‐Valle de Las Palmas) Mexicali Baja California México

2. Department of PathologyFederal University of Santa Catarina (UFSC) Florianópolis Brazil

3. Department of PathologyClinics Hospital University of São Paulo School of Medicine (HC‐FMUSP) São Paulo Brazil

4. Department of Oral Diagnosis, Oral PathologyPiracicaba Dental School, State University of Campinas (FOP‐UNICAMP) Piracicaba Brazil

5. Oral Pathology Section, Department of Stomatology, Public Oral Health, and Forensic DentistryUniversity of São Paulo, School of Dentistry of Ribeirão Preto (FORP/USP) Ribeirão Preto Brazil

6. Department of Pathology, Faculty of MedicineState University of Campinas (UNICAMP) Campinas Brazil

Funder

Coordenação de Aperfeiçoamento de Pessoal de Nível Superior

Fundação de Amparo à Pesquisa do Estado de São Paulo

Conselho Nacional de Desenvolvimento Científico e Tecnológico

Publisher

Wiley

Subject

Periodontics,Cancer Research,Otorhinolaryngology,Oral Surgery,Pathology and Forensic Medicine

Reference25 articles.

1. Fatty-acid synthase and human cancer: new perspectives on its role in tumor biology

2. Expression of fatty acid synthase (FASN) in oral nevi and melanoma

3. Enzymes of the fatty acid synthesis pathway are highly expressed in in situ breast carcinoma;Milgraum LZ;Clin Cancer Res,1997

4. Immunohistochemical expression of fatty acid synthase, apoptotic‐regulating genes, proliferating factors, and ras protein product in colorectal adenomas, carcinomas, and adjacent nonneoplastic mucosa;Visca P;Clin Cancer Res,1999

5. Pharmacological inhibitors of mammalian fatty acid synthase suppress DNA replication and induce apoptosis in tumor cell lines;Pizer ES;Cancer Res,1998

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线,采集、加工和组织学术论文而形成的新型学术文献查询和分析系统,可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容,当前同舟云学术共收录了国内外主流学术期刊6万余种,收集的期刊论文及会议论文总量共计约1.5亿篇,并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询!咨询电话:010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号  京ICP备18003416号-3