Alternative splicing regulation by Muscleblind proteins: from development to disease-Reference-Cited by-同舟云学术

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Alternative splicing regulation by Muscleblind proteins: from development to disease

Published:2011-04-13 Issue:4 Volume:86 Page:947-958
ISSN:1464-7931
Container-title:Biological Reviews
language:en
Short-container-title:

Author:

Fernandez-Costa Juan M.,Llamusi M. Beatriz,Garcia-Lopez Amparo,Artero Ruben

Publisher

Wiley

Subject

General Agricultural and Biological Sciences,General Biochemistry, Genetics and Molecular Biology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1469-185X.2011.00180.x/fullpdf

Reference110 articles.

1. RNA-dependent integrin alpha3 protein localization regulated by the Muscleblind-like protein MLP1.;Adereth;Nature Cell Biology,2005

2. A simple ligand that selectively targets CUG trinucleotide repeats and inhibits MBNL protein binding.;Arambula;Proceedings of the National Academy of Sciences of the United States of America,2009

3. The muscleblind gene participates in the organization of Z-bands and epidermal attachments of Drosophila muscles and is regulated by Dmef2.;Artero;Developmental Biology,1998

4. Partners in crime: bidirectional transcription in unstable microsatellite disease.;Batra;Human Molecular Genetics,2010

5. muscleblind, a gene required for photoreceptor differentiation in Drosophila, encodes novel nuclear Cys3His-type zinc-finger-containing proteins.;Begemann;Development,1997

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1. Alternative splicing and related RNA binding proteins in human health and disease;Signal Transduction and Targeted Therapy;2024-02-02

2. Msi2 enhances muscle dysfunction in a myotonic dystrophy type 1 mouse model;Biomedical Journal;2023-10

3. Muscleblind-1 interacts with tubulin mRNAs to regulate the microtubule cytoskeleton in C. elegans mechanosensory neurons;PLOS Genetics;2023-08-21

4. BlockmiR AONs as Site-Specific Therapeutic MBNL Modulation in Myotonic Dystrophy 2D and 3D Muscle Cells and HSALR Mice;Pharmaceutics;2023-03-31

5. Sustainable recovery of MBNL activity in autoregulatory feedback loop in myotonic dystrophy;Molecular Therapy - Nucleic Acids;2022-12

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