Changes in the Susceptibility of 12-O-Tetradecanoylphorbol 13-Acetate (TPA)-Treated HL-60 Cells to Staphylococcal Leukocidin

Author:

Morinaga Naoko1,Kato Iwao1,Noda Masatoshi1

Affiliation:

1. Second Department of Microbiology, School of Medicine; Chiba University; Chiba Chiba 260 Japan

Publisher

Wiley

Subject

Virology,Immunology,Microbiology

Reference19 articles.

1. Long-term phorbol ester treatment down-regulates protein kinase C and sensitizes the phosphoinositide signaling pathway to hormone and growth factor stimulation;Hepler;Evidence for a role of protein kinase C in agonist-induced desensitization. J. Biol. Chem.,1988

2. Action of staphylococcal toxins in cell cultures;Korbecki;J. Infect. Dis.,1965

3. Stimulation of Ca2+-dependent protein phosphorylation in HL-60 cells by staphylococcal leukocidin;Morinaga;FEMS Microbiol. Lett.,1987

4. Suppressive effect of calcium on the cytotoxicity of staphylococcal leukocidin for HL-60 cells;Morinaga;FEMS Microbiol. Lett.,1987

5. Changes in binding of staphylococcal leukocidin to HL-60 cells during differentiation induced by dimethyl sulfoxide;Morinaga;Infect. Immun.,1988

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