Insufficient Resistance of Trehalose-6,6′-Dimycolate-Treated T-Cell Receptor δ Gene Mutant (TCR δ-/-) Mice against Influenza Virus Infection

Author:

Hoq M. Mozammal1,Suzutani Tatsuo1,Nakaya Kazuhiro2,Yoshida Itsuro1,Ogasawara Masahiro1,Takeda Yoshinori1,Shibaki Taiichiro1,Itohara Shigeyoshi3,Yamamoto Hiroshi4,Azuma Masanobu1

Affiliation:

1. Department of Microbiology; Asahikawa Medical College; Asahikawa 078-8510 Japan

2. Animal Laboratory for Medical Research; Asahikawa Medical College; Asahikawa 078-8510 Japan

3. RIKEN Brain Science Institute; Wako Saitama 351-0198 Japan

4. Department of Immunology; Graduate School of Pharmaceutical Sciences, Osaka University; Suita Osaka 565-0871 Japan

Publisher

Wiley

Subject

Virology,Immunology,Microbiology

Reference17 articles.

1. Role of interferon in the augmented resistance of trehalose-6,6′-dimycolate-treated mice to influenza virus infection;Azuma;J. Gen. Virol.,1987

2. Correlation between augmented resistance to influenza virus infection and histological changes in lung of mice treated with trehalose-6,6′-dimycolate;Azuma;J. Biol. Response Mod.,1988

3. Blach-Olszewska , Z. Veckenstedt , A. Zaczynska , E. 1993 Does physiological interferon play a role in Mengo virus infection of mice ? Acta Virol. 37 68 72

4. Mice deficient in γδ T cells are resistant to lethal infection with Salmonella choleraesuis;Emoto;Infect. Immun.,1995

5. Effect of interferons and poly (I): poly (C) on the pathogenesis of the diabetogenic variant of encephalomyocarditis virus in different mouse strains;Giron;J. Interferon Res.,1988

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