Contribution of Anaphylatoxins to Allergic Inflammation in Human Lungs-Reference-Cited by-同舟云学术

Contribution of Anaphylatoxins to Allergic Inflammation in Human Lungs

Published:2005-11 Issue:11 Volume:49 Page:981-986
ISSN:0385-5600
Container-title:Microbiology and Immunology
language:en
Short-container-title:Microbiology and Immunology

Author:

Abe Masayoshi¹,Hama Hiroshi²,Shirakusa Takayuki³,Iwasaki Akinori³,Ono Nobuhumi²,Kimura Nobuhiro⁴,Hugli Tony E.⁵,Okada Noriko⁶,Katsuragi Takeshi¹,Okada Hidechika⁶

Affiliation:

1. Department of Pharmacology; Fukuoka University; Fukuoka Fukuoka 814-0180 Japan

2. Department of Drug Information, School of Pharmaceutical Science; Fukuoka University; Fukuoka Fukuoka 814-0180 Japan

3. Second Department of Surgery; Fukuoka University; Fukuoka Fukuoka 814-0180 Japan

4. First Department of Internal Medicine, School of Medicine; Fukuoka University; Fukuoka Fukuoka 814-0180 Japan

5. Torrey Pines Institute for Molecular Studies; San Diego CA92121 U.S.A.

6. Department of Biodefense Medicine; Nagoya City University Graduate School of Medical Sciences; Nagoya 467-8601 Japan

Publisher

Wiley

Subject

Virology,Immunology,Microbiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1348-0421.2005.tb03693.x/fullpdf

Reference22 articles.

1. Contribution of anaphylatoxin C5a to late airway responses after repeated exposure of antigen to allergic rats;Abe;J. Immunol.,2001

2. Molecular cloning and characterization of the human anaphylatoxin C3a receptor;Ames;J. Biol. Chem.,1996

3. Phramacological targeting of anaphylatoxin receptors during the effector phase of allergic asthma suppresses airway hyperresponsiveness and airway inflammation;Baelder;J. Immunol.,2005

4. Guinea pigs with a natural C3a-receptor defect exhibit decreased bronchoconstriction in allergic airway disease: evidence for an involvement of the C3a anaphylatoxin in the pathogenesis of asthma;Bautsch;J. Immunol.,2000

5. The orphan receptor C5L2 has high affinity binding sites for complement fragments C5a and C5a des-Arg;Cain;J. Biol. Chem.,2002

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