Recombinant human fibroblast growth factor and autogenous bone for periodontal regeneration: Alone or in combination? A randomized clinical trial

Author:

Kojima Kosuke1,Kamata Yohei1ORCID,Shimizu Tomoko1,Sato Satsuki1,Suzuki Sota1,Takanashi Yuya1,Hojo Sawako1,Yoshino Takeshi1,Fuchida Shinya2,Tamura Toshiyuki1,Minabe Masato3,Kodama Toshiro1,Kessoku Takaomi4,Oyamada Shunsuke5

Affiliation:

1. Department of Implantology and Periodontology, Graduate School of Dentistry Kanagawa Dental University Yokohama Kanagawa Japan

2. Department of Education Planning Kanagawa Dental University Yokosuka Japan

3. Bunkyodori Dental Clinic Chiba Japan

4. Department of Gastroenterology International University Health and Welfare Graduate School of Medicine Chiba Japan

5. Department of Biostatistics JORTC Data Center 2‐54‐6‐302 Tokyo Japan

Abstract

AbstractAimTo compare the outcomes of therapy using recombinant human fibroblast growth factor (rhFGF)‐2 combined with autologous bone grafting (ABG) therapy with those of rhFGF‐2 alone and ABG alone in the treatment of periodontal intraosseous defects.MethodsPeriodontal intraosseous defects were randomized to receive rhFGF‐2 therapy + ABG, rhFGF‐2 therapy alone, or ABG alone. Periodontal examination and periapical radiography were performed preoperatively and at 3, 6, and 12 months postoperatively.ResultsAt the 12 months follow‐up, all three groups showed significant improvement in the clinical attachment level (CAL): 5.6 ± 1.6, 5.8 ± 1.7, and 5.2 ± 1.6 mm in the rhFGF‐2 + ABG, rhFGF‐2 alone, and ABG alone groups, respectively, with no significant inter‐group differences (p < .05). rhFGF‐2 therapy (alone or in combination) resulted in greater bone defect filling (BDF) (2.3 ± 1.2 mm and 2.6 ± 1.9 mm, respectively) than ABG therapy alone (1.2 ± 1.2 mm). Gingival recession was lesser in the ABG alone (1.2 ± 1.1 mm) and rhFGF‐2 + ABG groups (1.4 ± 0.8 mm) than in the rhFGF‐2 alone group (2.2 ± 1.2 mm).ConclusionThe results of this study showed that at 12 months postoperatively, all treatments resulted in statistically significant clinical improvements compared to the baseline. From these results, it can be concluded that rhFGF‐2 promotes hard tissue regeneration in intraosseous defects.

Publisher

Wiley

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