Acrogeria with Decreased Gene Expression of α1(I) and α1(III) Collagen in Cultured Dermal Fibroblasts
Author:
Affiliation:
1. Department of Molecular Genetics, College of Medicine; University of Cincinnati; Cincinnati OH 45267 USA
2. Department of Dermatology; Dokkyo University of Medicine; Tochigi Japan
Publisher
Wiley
Subject
Dermatology,General Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1346-8138.2004.tb00550.x/fullpdf
Reference19 articles.
1. Familial acrogeria (Gottron);Groot;Br J Dermatol,1980
2. Human skin collagen. Presence of type I and type III at all levels of the dermis;Epstein;J Biol Chem,1978
3. Similar, but not identical, modulation of expression of extracellular matrix components during in vitro and in vivo aging of human skin fibroblasts;Takeda;J Cell Physiol,1992
4. The cell biology of aging;Hayflich;J Invest Dermatol,1979
5. Reduced expression of mRNA for transforming growth factor β (TGFβ) and TGFβ receptors I and II and decreased TGFβ binding to the receptors in in vitro-aged fibroblasts;Mori;Arch Dermatol Res,1998
Cited by 2 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Acrogeria: A Rare Congenital Aging Syndrome;Indian Journal of Paediatric Dermatology;2023-10
2. The collαgen III fibril has a “flexi-rod” structure of flexible sequences interspersed with rigid bioactive domains including two with hemostatic roles;PLOS ONE;2017-07-13
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