Impaired Contact Hypersensitivity Reaction and Reduced Production of Vascular Endothelial Growth Factor in Tumor Necrosis Factor-α Gene-Deficient Mice-Reference-Cited by-同舟云学术

Impaired Contact Hypersensitivity Reaction and Reduced Production of Vascular Endothelial Growth Factor in Tumor Necrosis Factor-α Gene-Deficient Mice

Published:2005-07 Issue:7 Volume:32 Page:523-533
ISSN:0385-2407
Container-title:The Journal of Dermatology
language:en
Short-container-title:The Journal of Dermatology

Author:

Shibata Mari¹,Sueki Hirohiko¹,Suzuki Hirotake¹,Watanabe Hideaki¹,Ohtaki Hirokazu²,Shioda Seiji²,Nakanishi-Ueda Takako³,Yasuhara Hajime³,Sekikawa Kenji⁴,Iijima Masafumi¹

Affiliation:

1. Department of Dermatology; Showa University School of Medicine; Tokyo Japan

2. Department of First Anatomy; Showa University School of Medicine; Tokyo Japan

3. Department of Second Pharmacology; Showa University School of Medicine; Tokyo Japan

4. Division of Immunology; National Institute of Animal Health; Kannondai 3-1-1 Tsukuba Ibaraki 305-0856 Japan

Publisher

Wiley

Subject

Dermatology,General Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1346-8138.2005.tb00794.x/fullpdf

Reference48 articles.

1. Tumor necrosis factor is a critical mediator in hapten-induced irritant and contact hypersensitivity reactions;Piguet;J Exp Med,1991

2. Dermal tumor necrosis factor-α induces dendritic cell migration to draining lymph nodes, and possibly provides one stimulus for Langerhans cell migration;Cumberbatch;Immunology,1992

3. Cis-urocanic acid suppression of contact hypersensitivity induction is mediated via tumor necrosis factor-α;Kurimoto;J Immunol,1992

4. Sunlight and skin-associated lymphoid tissues (SALT): If UVB is the trigger and TNF α is its mediator, what is the message?;Streilein;J Invest Dermatol,1993

5. Tumor necrosis factor-α is required for accumulation of dendritic cells in draining lymph nodes and for optimal contact sensitization;Cumberbatch;Immunology,1995

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