AKT‐dependent hyperproliferation of keratinocytes in familial hidradenitis suppurativa with a NCSTN mutation: a potential role of defective miR‐100‐5p
Author:
Affiliation:
1. Institute of Dermatology Jiangsu Key Laboratory of Molecular Biology for Skin Diseases and STIs Chinese Academy of Medical Sciences and Peking Union Medical College Nanjing Jiangsu 210042 China
Funder
National Natural Science Foundation of China
Publisher
Wiley
Subject
Dermatology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/bjd.18460
Reference8 articles.
1. Hidradenitis suppurativa: A comprehensive review
2. Mutations in the γ-Secretase Genes NCSTN , PSENEN , and PSEN1 Underlie Rare Forms of Hidradenitis Suppurativa (Acne Inversa)
3. Genetic analysis of NCSTN for potential association with hidradenitis suppurativa in familial and nonfamilial patients
4. Nicastrin mutations in familial acne inversa impact keratinocyte proliferation and differentiation through the Notch and phosphoinositide 3-kinase/AKT signalling pathways
5. MicroRNAs as biological regulators in skin disorders
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4. Discovery and Potential Functional Characterization of Long Noncoding RNAs Associated with Familial Acne Inversa with NCSTN Mutation;Dermatology;2023-07-25
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