Haematological effects of oral administration of bitopertin, a glycine transport inhibitor, in patients with non‐transfusion‐dependent β‐thalassaemia-Reference-Cited by-同舟云学术

Haematological effects of oral administration of bitopertin, a glycine transport inhibitor, in patients with non‐transfusion‐dependent β‐thalassaemia

Published:2021-04-30 Issue:2 Volume:194 Page:474-477
ISSN:0007-1048
Container-title:British Journal of Haematology
language:en
Short-container-title:Br. J. Haematol.

Author:

Taher Ali T.¹^ORCID,Viprakasit Vip²,Cappellini Maria Domenica³^ORCID,Kraus Dominik⁴,Cech Patrick⁴,Volz Dietmar⁴,Winter Erica⁵,Nave Stephane⁴,Dukart Juergen⁴⁶⁷,Khwaja Omar⁴,Koerner Annette⁴,Hermosilla Ricardo⁴,Brugnara Carlo⁸⁹^ORCID

Affiliation:

1. Department of Internal Medicine Division of Hematology and Oncology American University of Beirut Medical Center Beirut Lebanon

2. Department of Pediatrics and Siriraj Thalassemia Center Division of Hematology and Oncology Mahidol University Bangkok Thailand

3. Department of Medicine Ca'Granda Foundation IRCCS University of Milan Milan Italy

4. Roche Pharma Research and Early Development Roche Innovation Center Basel Basel Switzerland

5. Roche Pharma Research and Early Development Roche Innovation Center New York New York NY USA

6. Institute of Neuroscience and Medicine Brain & Behaviour (INM‐7) Research Centre Jülich JülichGermany

7. Institute of Systems Neuroscience Medical Faculty Heinrich Heine University Düsseldorf Düsseldorf Germany

8. Department of Laboratory Medicine Boston Children’s Hospital Boston MAUSA

9. Department of Pathology Harvard Medical School Boston MA USA

Publisher

Wiley

Subject

Hematology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/bjh.17479

Reference9 articles.

1. Effects of GlyT1 inhibition on erythropoiesis and iron homeostasis in rats

2. Glycine transport by human red blood cells and ghosts: evidence for glycine anion and proton cotransport by band 3

3. Glycine transport in human erythrocytes.

4. -thalassemias: paradigmatic diseases for scientific discoveries and development of innovative therapies

5. What can we learn from ineffective erythropoiesis in thalassemia?

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