Does the FSHB c.‐211G>T polymorphism impact Sertoli cell number and the spermatogenic potential in infertile patients?

Author:

Schubert Maria1,Kaldewey Sophie1,Pérez Lanuza Lina2,Krenz Henrike3,Dugas Martin3,Berres Sven2,Kliesch Sabine1,Wistuba Joachim2,Gromoll Jörg2ORCID

Affiliation:

1. Department of Clinical and Surgical Andrology Centre of Reproductive Medicine and Andrology University of Münster Münster Germany

2. Institute of Reproductive and Regenerative Biology Centre of Reproductive Medicine and Andrology University of Münster Münster Germany

3. Institute of Medical Informatics—Informatics for Personalized Medicine University of Münster Münster Germany

Funder

Deutsche Forschungsgemeinschaft

Publisher

Wiley

Subject

Urology,Endocrinology,Reproductive Medicine,Endocrinology, Diabetes and Metabolism

Reference35 articles.

1. Diagnosis of Male Infertility: Diagnostic Work-up of the Infertile Man

2. Disorders of spermatogenesis: Perspectives for novel genetic diagnostics after 20 years of unchanged routine;Tüttelmann F;Med Genet,2018

3. Pharmacogenetics of FSH Action in the Male

4. A short evolutionary history of FSH‐stimulated spermatogenesis;Huhtaniemi I;Hormones (Athens),2015

5. Evidence From Sertoli Cell-Depleted Rats Indicates That Spermatid Number in Adults Depends on Numbers of Sertoli Cells Produced During Perinatal Development*

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