Feedback suppression of meal-induced glucagon-like peptide-1 (GLP-1) secretion mediated through elevations in intact GLP-1 caused by dipeptidyl peptidase-4 inhibition: a randomized, prospective comparison of sitagliptin and vildagliptin treatment-Reference-Cited by-同舟云学术

Feedback suppression of meal-induced glucagon-like peptide-1 (GLP-1) secretion mediated through elevations in intact GLP-1 caused by dipeptidyl peptidase-4 inhibition: a randomized, prospective comparison of sitagliptin and vildagliptin treatment

Published:2016-08-17 Issue:11 Volume:18 Page:1100-1109
ISSN:1462-8902
Container-title:Diabetes, Obesity and Metabolism
language:en
Short-container-title:Diabetes Obes Metab

Author:

Baranov Oleg¹,Kahle Melanie¹²,Deacon Carolyn F.³,Holst Jens J.³,Nauck Michael A.¹²

Affiliation:

1. Diabeteszentrum Bad Lauterberg; Bad Lauterberg im Harz Germany

2. Division of Diabetology, Medical Department I, St. Josef-Hospital; Ruhr-University Bochum; Bochum Germany

3. Department of Biomedical Sciences, Panum Institute; University of Copenhagen; Copenhagen Denmark

Funder

Novartis Pharma

Publisher

Wiley

Subject

Endocrinology,Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/dom.12706/fullpdf

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2. Incretin therapies-highlighting common features and differences in the modes of action of GLP-1 receptor agonists and DPP-4 inhibitors;Nauck;Diabetes Obes Metab,2016

3. Glycaemic efficacy of glucagon-like peptide-1 receptor agonists and dipeptidyl peptidase-4 inhibitors as add-on therapy to metformin in subjects with type 2 diabetes-a review and meta analysis;Deacon;Diabetes Obes Metab,2012

4. Dipeptidyl-peptidase IV hydrolyses gastric inhibitory polypeptide, glucagon-like peptide-1(7-36)amide, peptide histidine methionine and is responsible for their degradation in human serum;Mentlein;Eur J Biochem/FEBS,1993

5. Degradation of glucose-dependent insulinotropic polypeptide and truncated glucagon-like peptide 1 in vitro and in vivo by dipeptidyl peptidase IV;Kieffer;Endocrinology,1995

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