Monomeric C‐reactive protein via endothelial CD31 for neurovascular inflammation in an ApoE genotype‐dependent pattern: A risk factor for Alzheimer’s disease?-Reference-Cited by-同舟云学术

Monomeric C‐reactive protein via endothelial CD31 for neurovascular inflammation in an ApoE genotype‐dependent pattern: A risk factor for Alzheimer’s disease?

Published:2021-10-23 Issue:11 Volume:20 Page:
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Zhang Zhengrong¹^ORCID,Na Hana¹,Gan Qini¹,Tao Qiushan¹,Alekseyev Yuriy²,Hu Junming³,Yan Zili¹,Yang Jack B.¹,Tian Hua¹⁴,Zhu Shenyu¹,Li Qiang¹⁵,Rajab Ibraheem M.⁶,Blusztajn Jan Krizysztof⁷,Wolozin Benjamin¹,Emili Andrew⁸,Zhang Xiaoling³,Stein Thor⁷⁹¹⁰¹¹,Potempa Lawrence A.⁶,Qiu Wei Qiao¹⁹¹²

Affiliation:

1. Department of Pharmacology and Experimental Therapeutics Boston University School of Medicine Boston Massachusetts USA

2. Microarray and Sequencing Core Facility Boston University School of Medicine Boston Massachusetts USA

3. Department of Medicine Boston University School of Medicine Boston Massachusetts USA

4. Department of Pharmacology Xiaman Medical College Xiaman China

5. Nursing School Qiqihar Medical University Qiqihar China

6. Roosevelt University College of Pharmacy Schaumburg Illinois USA

7. Department of Pathology and Laboratory Medicine Boston University School of Medicine Boston Massachusetts USA

8. Department of Biochemistry Boston University School of Medicine Boston Massachusetts USA

9. Alzheimer’s Disease Center Boston University School of Medicine Boston Massachusetts USA

10. VA Boston Healthcare System Boston Massachusetts USA

11. Department of Veterans Affairs Medical Center Bedford Massachusetts USA

12. Department of Psychiatry Boston University School of Medicine Boston Massachusetts USA

Funder

National Institute of Neurological Disorders and Stroke

National Heart, Lung, and Blood Institute

National Institute on Aging

Publisher

Wiley

Subject

Cell Biology,Aging

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.13501

Reference63 articles.

1. Prenatal Exposure to apoE Deficiency and Postnatal Hypercholesterolemia Are Associated with Altered Cell-Specific Lysine Methyltransferase and Histone Methylation Patterns in the Vasculature

2. Apolipoprotein E controls cerebrovascular integrity via cyclophilin A

3. Reconstruction of the human blood–brain barrier in vitro reveals a pathogenic mechanism of APOE4 in pericytes

4. Monomeric C-reactive protein and Notch-3 co-operatively increase angiogenesis through PI3K signalling pathway

5. Mitochondrial Mechanisms in Cerebral Vascular Control: Shared Signaling Pathways with Preconditioning

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