Microglial cathepsin E plays a role in neuroinflammation and amyloid β production in Alzheimer’s disease-Reference-Cited by-同舟云学术

Microglial cathepsin E plays a role in neuroinflammation and amyloid β production in Alzheimer’s disease

Published:2022-02-19 Issue:3 Volume:21 Page:
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Xie Zhen¹,Meng Jie²³,Kong Wei¹^ORCID,Wu Zhou³,Lan Fei¹,Narengaowa ¹,Hayashi Yoshinori⁴,Yang Qinghu⁵,Bai Zhantao⁵,Nakanishi Hiroshi⁶,Qing Hong¹,Ni Junjun¹³^ORCID

Affiliation:

1. Key Laboratory of Molecular Medicine and Biotherapy Department of Biology School of Life Science Beijing Institute of Technology Beijing China

2. Department of Neurology and State Key Laboratory of Biotherapy Collaborative Innovation Center for Biotherapy West China Hospital Sichuan University Chengdu China

3. Department of Aging Science and Pharmacology Faculty of Dental Science Kyushu University Fukuoka Japan

4. Department of Physiology Nihon University School of Dentistry Tokyo Japan

5. Research Center for Resource Peptide Drugs Shaanxi Engineering & Technological Research Center for Conversation & Utilization of Regional Biological Resources Yan’an University Yan’an China

6. Department of Pharmacology Faculty of Pharmacy Yasuda Women’s University Hiroshima Japan

Publisher

Wiley

Subject

Cell Biology,Aging

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.13565

Reference45 articles.

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2. Clearance of senescent glial cells prevents tau-dependent pathology and cognitive decline

3. Neutralization of TNFSF10 ameliorates functional outcome in a murine model of Alzheimer’s disease

4. Neutralization of TRAIL death pathway protects human neuronal cell line from β-amyloid toxicity

5. BACE1 is at the crossroad of a toxic vicious cycle involving cellular stress and β-amyloid production in Alzheimer’s disease

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