Accumbal and pallidal dopamine, glutamate and GABA overflow during cocaine self-administration and its extinction in rats-Reference-Cited by-同舟云学术

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Accumbal and pallidal dopamine, glutamate and GABA overflow during cocaine self-administration and its extinction in rats

Published:2013-01-14 Issue:2 Volume:18 Page:307-324
ISSN:1355-6215
Container-title:Addiction Biology
language:en
Short-container-title:

Author:

Wydra Karolina¹,Golembiowska Krystyna²,Zaniewska Magdalena¹,Kamińska Katarzyna²,Ferraro Luca³,Fuxe Kjell⁴,Filip Małgorzata

Affiliation:

1. Laboratory of Drug Addiction Pharmacology; Department of Pharmacology; Institute of Pharmacology Polish Academy of Sciences; Poland

2. Department of Pharmacology; Institute of Pharmacology Polish Academy of Sciences; Poland

3. Department of Life Sciences and Biotechnology; LTTA Centre; University of Ferrara; Italy

4. Department of Neuroscience; Karolinska Institutet; Sweden

Publisher

Wiley

Subject

Psychiatry and Mental health,Pharmacology,Medicine (miscellaneous)

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/adb.12031/fullpdf

Reference55 articles.

1. Nonvesicular release of neurotransmitter;Attwell;Neuron,1993

2. N-acetyl cysteine-induced blockade of cocaine-induced reinstatement;Baker;Ann N Y Acad Sci,2003

3. Cystine/glutamate exchange serves as the source for extracellular glutamate: modifications by repeated cocaine administration;Baker;Amino Acids,2002

4. Effect of GABA agonists and GABA-A receptor modulators on cocaine- and food-maintained responding and cocaine discrimination in rats;Barrett;J Pharmacol Exp Ther,2005

5. Context-specific enhancement of glutamate transmission by cocaine;Bell;Neuropsychopharmacology,2000

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