Morphine treatment enhances glutamatergic input onto neurons of the nucleus accumbens via both disinhibitory and stimulating effect-Reference-Cited by-同舟云学术

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Morphine treatment enhances glutamatergic input onto neurons of the nucleus accumbens via both disinhibitory and stimulating effect

Published:2016-08-22 Issue:6 Volume:22 Page:1756-1767
ISSN:1355-6215
Container-title:Addiction Biology
language:en
Short-container-title:Addiction Biology

Author:

Yuan Kejing¹,Sheng Huan¹,Song Jiaojiao¹,Yang Li¹,Cui Dongyang¹,Ma Qianqian¹,Zhang Wen¹,Lai Bin¹,Chen Ming¹,Zheng Ping¹

Affiliation:

1. State Key Laboratory of Medical Neurobiology, School of Basic Medical Sciences and Institutes of Brain Science; Fudan University; China

Funder

National Program of Basic Research sponsored by the Ministry of Science and Technology of China

Science and Technology Program of Yunnan Province

Project of Foundation of National Natural Science of China

Publisher

Wiley

Subject

Psychiatry and Mental health,Pharmacology,Medicine (miscellaneous)

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/adb.12438/fullpdf

Reference31 articles.

1. Orexin A in the VTA is critical for the induction of synaptic plasticity and behavioral sensitization to cocaine;Borgland;Neuron,2006

2. Morphine disinhibits glutamatergic input to VTA dopamine neurons and promotes dopamine neuron excitation;Chen;eLife,2015

3. Dopaminergic regulation of limbic-striatal interplay;Floresco;J Psychiatry Neurosci,2007

4. Excitatory effects of dopamine released by impulse flow in the rat nucleus accumbens in vivo;Gonon;Neuroscience,1996

5. Endogenous and exogenous dopamine depress EPSCs in rat nucleus accumbens in vitro via D1 receptors activation;Harvey;J Physiol,1996

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