Neuroinfection may contribute to pathophysiology and clinical manifestations of COVID‐19-Reference-Cited by-同舟云学术

Neuroinfection may contribute to pathophysiology and clinical manifestations of COVID‐19

Published:2020-04-11 Issue:3 Volume:229 Page:
ISSN:1748-1708
Container-title:Acta Physiologica
language:en
Short-container-title:Acta Physiol

Author:

Steardo Luca¹²,Steardo Luca³,Zorec Robert⁴⁵^ORCID,Verkhratsky Alexei⁶⁷^ORCID

Affiliation:

1. Fortunato University Benevento Italy

2. Sapienza University Rome Italy

3. University Magna Graecia Catanzaro Italy

4. Clica BIOMEDICAL Ljubljana Slovenia

5. Laboratory of Neuroendocrinology‐Molecular Cell Physiology Institute of Pathophysiology Faculty of Medicine University of Ljubljana Ljubljana Slovenia

6. Faculty of Biology, Medicine and Health The University of Manchester Manchester UK

7. Achucarro Center for Neuroscience IKERBASQUE Bilbao Spain

Publisher

Wiley

Subject

Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/apha.13473

Reference29 articles.

1. Epidemiology and Clinical Presentations of the Four Human Coronaviruses 229E, HKU1, NL63, and OC43 Detected over 3 Years Using a Novel Multiplex Real-Time PCR Method

2. Response to Commentary on “The neuroinvasive potential of SARS‐CoV‐2 may play a role in the respiratory failure of COVID‐19 patients”

3. Axonal Transport Enables Neuron-to-Neuron Propagation of Human Coronavirus OC43

4. Neurotropic virus tracing suggests a membranous-coating-mediated mechanism for transsynaptic communication

5. Neurotropism of Human Coronavirus 229E

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