A point mutation c.473A > G of ITGB3 is responsible for the formation of the Wo a human platelet alloantigen-Reference-Cited by-同舟云学术

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A point mutation c.473A > G of ITGB3 is responsible for the formation of the Wo a human platelet alloantigen

Published:2019-12-20 Issue:1 Volume:60 Page:
ISSN:0041-1132
Container-title:Transfusion
language:en
Short-container-title:Transfusion

Author:

Holzwarth Sarah T.¹,Strobel Julian²,Cooper Nina¹³,Leyh Jörg⁴,Bayat Behnaz¹,Bein Gregor¹³,Zingsem Jürgen²,Sachs Ulrich J.¹³^ORCID

Affiliation:

1. Institute for Clinical Immunology and Transfusion MedicineJustus Liebig University Giessen Germany

2. Department of Transfusion Medicine and HaemostaseologyFriedrich‐Alexander‐University Erlangen Germany

3. German Center for Feto‐Maternal Incompatibility (DZFI)University Hospital Giessen and Marburg Giessen Germany

4. Children's HospitalFriedrich‐Alexander‐University Erlangen Germany

Publisher

Wiley

Subject

Hematology,Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/trf.15640

Reference4 articles.

1. New low-frequency platelet glycoprotein polymorphisms associated with neonatal alloimmune thrombocytopenia

2. Alloantibody against new platelet alloantigen (Lapa) on glycoprotein IIb is responsible for a case of fetal and neonatal alloimmune thrombocytopenia

3. Monoclonal antibody--specific immobilization of platelet antigens (MAIPA): a new tool for the identification of platelet-reactive antibodies

4. Reference SNP (rs) report for rs748901429. [cited September 26 2019].https://www.ncbi.nlm.nih.gov/snp/rs748901429#frequency_tab).

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Fetale und neonatale Alloimmunthrombozytopenie, Teil 1;Transfusionsmedizin;2021-05

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